"no more embarrassing trips to the doctor's office for me."

happy couple

“ seven years worry free for me thanks to HERBASTAT
...Oh and my partner is HERPES FREE too!!! “


"HERBASTAT's discreet packaging means my friends and neighbors will never know."

Ok bad luck for you

YOU have herpes ……What are you going to do?

Pray that you don’t get another outbreak……hope it never returns…… OR are you going to give your body what it needs to fight off an invasion?

Your immune system is your body's evolved weapon for defending itself against micro-pathogens, such as the Herpes virus. Your immune system may not be able to completely eliminate herpes but it is your body's first line of defense.

Herbastat makes the immune system colony speak the same language. This is what makes Herbastat so different from any other drug or herbal remedy. Your Immune system is very complex and each department has a specific agenda and they act separately from one another Herbastat causes the Immune system to have the same agenda.

It's no secret that the earth has every herb on the planet to cure our illnesses. The problem is finding the herb or combination of herbs and vitamins to cure your illness. Herbastat has the combination of Sugars, herbs, vitamins and minerals to stop the symptoms of Herpes. All you have to do is add a teaspoon of this to your daily smoothie. The average daily diet will never give you the type of immune system that will fight off a common cold much less fight off a herpes outbreak.

Below we will explain in a more technical fashion.

Immunostimulants, also known as immunostimulators, are substances (drugs and nutrients) that stimulate the immune system by inducing activation or increasing activity of any of its components. One notable example is the granulocyte macrophage colony-stimulating factor. The herpes virus attacks when we are under stress and our immune system is low. Your herpes virus strikes when are immune system is deprived. Its your immune system that allows the flare up.

Herbastat super charges your Granulocyte Macrophage Colony,
what's that you ask?

Here are the main player's: neutrophils, macrophages, killer cells, T and B cells. Granulocyte macrophage Colony (GM-CSF) is a cytokine that functions as a white blood cell growth factor. GM-CSF stimulates stem cells to produce granulocytes (neutrophils, eosinophils, and basophils) and monocytes. Monocytes exit the circulation and migrate into tissue, whereupon they mature into macrophages. It is thus part of the immune/inflammatory cascade, by which activation of a small number of macrophages can rapidly lead to an increase in their numbers, a process crucial for fighting infection.

Herbastat turbo boosts the cells that fight invasion.

The Killer cells

Given their strong cytolytic activity and the potential for auto-reactivity, NK cell activity is tightly regulated. NK cells must receive an activating signal, which can come in a variety of forms, the most important of which are listed below.

NK cells are cytotoxic; small granules in their cytoplasm contain proteins such as perforin and proteases known as granzymes. Upon release in close proximity to a cell slated for killing, perforin forms pores in the cell membrane of the target cell, creating an aqueous channel through which the granzymes and associated molecules can enter, inducing either apoptosis or osmotic cell lysis. The distinction between apoptosis and cell lysis is important in immunology: lysing a virus-infected cell would only release the virions, whereas apoptosis leads to destruction of the virus inside.
NK cells are activated in response to interferons or macrophage-derived cytokines. They serve to contain viral infections while the adaptive immune response is generating antigen-specific cytotoxic T cells that can clear the infection.
Patients deficient in NK cells prove to be highly susceptible to early phases of herpes virus infection.
In order for NK cells to defend the body against viruses and other pathogens, they require mechanisms that enable the determination of whether a cell is infected or not. The exact mechanisms remain the subject of current investigation, but recognition of an "altered self" state is thought to be involved. To control their cytotoxic activity, NK cells possess two types of surface receptors: activating receptors and inhibitory receptors. Most of these receptors are not unique to NK cells and can be present in some T cell subsets as well.

These inhibitory receptors recognize MHC class I alleles, which could explain why NK cells kill cells possessing low levels of MHC class I molecules. This inhibition is crucial to the role played by NK cells. MHC class I molecules consist of the main mechanism by which cells display viral or tumor antigens to cytotoxic T-cells. A common evolutionary adaption to this seen in both intracellular microbes and tumours is a chronic down-regulation of these MHC I molecules, rendering the cell impervious to T-cell mediated immunity. It is believed that NK cells, in turn, evolved as an evolutionary response to this adaption, as the loss of the MHC would deprive these cells of the inhibitory effect of MHC and render these cells vulnerable to NK cell mediated apoptosis.

Function of T helper cells: Antigen presenting cells (APCs) present antigen on their Class II MHC molecules (MHC2). Helper T cells recognize these, with the help of their expression of CD4 co-receptor (CD4+). The activation of a resting helper T cell causes it to release cytokines and other stimulatory signals (green arrows) that stimulate the activity of macrophages, killer T cells and B cells, the latter producing antibodies.

T helper cells (also known as Th cells) are a sub-group of lymphocytes (a type of white blood cell or leukocyte) that play an important role in establishing and maximizing the capabilities of the immune system. These cells are unusual in that they have no cytotoxic or phagocytic activity; they cannot kill infected host cells (also known as somatic cells) or pathogens, and without other immune cells they would usually be considered useless against an infection. The cells are involved in activating and directing other immune cells, and are particularly important in the immune system. They are essential in determining B cell antibody class switching, in the activation and growth of cytotoxic T cells, and in maximizing bactericidal activity of phagocytes such as macrophages. It is this diversity in function and their role in influencing other cells that gives T helper cells their name.

Mature T cells are believed to always express the surface protein CD4. T cells expressing CD4 are also known as CD4+ T cells. CD4+ T cells are generally treated as having a pre-defined role as helper T cells within the immune system, although there are known rare exceptions. For example, there are sub-groups of regulatory T cells, natural killer T cells, and cytotoxic T cells that are known to express CD4 (although cytotoxic examples have been observed in extremely low numbers in specific disease states, they are usually considered non-existent). All of the latter CD4+ T cell groups are not considered T helper cells. Herbastat causes your complex Immune system to speak the same language. This way they work with a more common goal. In your case stop a HSV breakout.